The problem with using any anacotes of seeing people suffering from one addiction automatically suffering from another addiction is that it is sensational in matter and limited, also. Selection bias, essentially. Not only are you looking for cases of addiction, but the examples of addiction will be more obvious. Are you likely to reference or even be aware of the person getting by in society? No. They will not come to your attention unless they desperately want to be acknowledged, which most addicts do not. Furthermore, I have heard people flat out not believe their stories of addiction since it did not fit the idea of addiction held by the observer.

As for whether or not alcohol is a depressant and what being a depressant means... I did not say that alcohol does not contribute to depression (or it is not possible for alcohol to contribute to depression). I said that being in the drug class of depressant does not mean it causes psychological depression.

I realize that wikipedia might not be the best source, but every scientific source tends to agree with wikipedia (in this case) and feel free to google any one of them...

A depressant, or central depressant, is a drug or endogenous compound that lowers neurotransmission levels, which is to depress or reduce arousal or stimulation, in various areas of the brain.[1] Depressants are also occasionally referred to as "downers" as they lower the level of arousal when taken. Stimulants or "uppers" increase mental and/or physical function and are the functional opposites of depressants.
Depressants are widely used throughout the world as prescription medicines and as illicit substances. When these are used, effects often include ataxia, anxiolysis, pain relief, sedation or somnolence, and cognitive/memory impairment, as well as in some instances euphoria, dissociation, muscle relaxation, lowered blood pressure or heart rate, respiratory depression, and anticonvulsant effects, and even complete anesthesia or death at high doses.
Depressants exert their effects through a number of different pharmacological mechanisms, the most prominent of which include facilitation of GABA, and inhibition of glutamatergic or monoaminergic activity. Other examples are chemicals that modify the electrical signaling inside the body. The most prominent of these being bromides and channel blockers.

I didnt bother googling further than that since it is not relevant to the discussion. Whether or not it is a depressant or stimulant, itself, is irrelevant to the argument. Many psych meds are either depressants or stimulants... and since depressants and stimulants are the opposites of each other, if one were bad, the other would be good.... A depressant does not mean it causes depression, this was a red herring, that was why I brought it up.